My research is focused on understanding the mechanisms of information flow in signal transductionnetworks controlling cell growth, survival, and metabolism. At UC Davis, my research group uses singlecell techniques to track protein and gene expression activity simultaneously in single cells, and we use these data to build quantitative models that predict the responses of healthy and diseased cells to drugs. I joined UC Davis as an Assistant Professor in 2013, following postdoctoral training under Joan Brugge at Harvard Medical School. My work there was funded by a Department of Defense Multidisciplinary Postdoctoral Fellowship and focused on the role of ERK and Akt in controlling cell proliferation in breast cancer. Prior to that, I obtained my Ph.D. in Computational Systems Biology from MIT, studying Death Receptor signaling in the lab of Peter Sorger. My undergraduate work was done at Cornell University, where I studied the regulation of Cdc42 in the lab of Rick Cerione